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Mittwoch, 16. September 2015, 10:59

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Western blot for P ERK1 2 and total ERK1 o levels revealed
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tivation upon sorting of LRCs that have been slow cycling or arrested in the
tumor. We conclude that in LRCs, the transient growth arrest and low ERK1 2
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ERK1 2 tivity that correlates with tive proliferation. The rmation above rend
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response to an unidentified signal that tivates their G0 G1 exit in vivo. HEp3
NLRCs are also poised for developnt,but apparently must undergo a reprogramming
phase that requires longer. A similar situation was reported for So Headhe
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